digital slide 发布。这是将数字化切片嵌入weblecture幻灯的位置，实现多人共览数字化切片并能在线交流。全球首发。

weblecture发布1.282版，这是exe版的程序。因为使用了air的交互（与本地的程序交互）。实际为air的打包  adt -package -target native  weblecture.exe weblecture.air

程序实现录音到桌面，生成mp3 。

详细请看  http://www.ccp.net.cn/weblecture  

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WebLecture 添加录音功能，即将正式发布

新教室即将加入本地录音功能，录音mic，另存到本地。
需要flash10.1   地址在这里flashplayer10_1.rar

测试在这里  http://www.ccp.net.cn/classroom/3.html

添加评论COMMENT 系统
增加互动性。

根据我的测试，目前最稳定的浏览器，IE8

我曾经测试打开30个浏览页面，可以稳定的浏览。

这可不是广告。

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流氓的新生代，无耻的下载站
今天看到在完美者论坛看到这样描写下载站。
我现在很少用国内的下载网站了，需要的软件都来自rapidshare.

最近在做电子书，程序等。感觉这些话写出了我的最真实的感受。

New Delhi Metallo-beta-lactamase (NDM-1)^[1] is a newly-identified enzyme that makes bacteria resistant to broad range of beta-lactam antibiotics. This includes antibiotics of the carbapenem family that were a mainstay for the treatment of other antibiotic-resistant bacteria. The gene for NDM-1 is one member of a large gene family that encode beta-lactamase enzymes called carbapenemases. Bacteria that carry such genes are often referred to in the news media as "superbugs", since infections with these bacteria are very hard to treat successfully. Indeed, the United Kingdom Health Protection Agency has stated that "most isolates with NDM-1 enzyme are resistant to all standard intravenous antibiotics for treatment of severe infections."^[2]

NDM-1 was first identified in December 2009 in a patient who was hospitalised in New Delhi with a Klebsiella pneumoniae infection. It has since been detected in bacteria in India, Pakistan, the United Kingdom and the United States. The most common organisms expressing this enzyme are the Gram-negative bacteria Escherichia coli and Klebsiella pneumoniae, but the gene for NDM-1 can be transmitted from one strain of bacteria to another through horizontal gene transfer. The emergence of resistant Gram-negative bacteria is in contrast to the more familiar antibiotic resistant bacteria such as Methicillin-resistant Staphylococcus aureus (MRSA), which are Gram-positive.

Carbapenems are a class of beta-lactam antibiotics were, until recently, capable of killing most bacteria by inhibiting the synthesis of one of their cell wall layers. The carbapenems were developed to be less sensitive to inactivation by the beta-lactamase enzymes that had evolved in bacteria resistant to antibiotics such as penicillin. The bla_NDM-1 gene produces NDM-1, which is a carbapenemase beta-lactamase - an enzyme that hydrolyzes and inactivates these carbapenem antibiotics.

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